Our Greatest Accomplishment: Grass-Roots Action Has Forced a Major Shift in Thinking

Our Greatest Accomplishment:
Grass-Roots Action Has Forced a Major Shift in Thinking

[This article, with some of the footnotes removed, appeared in
The Workbook [Southwest Research and Information Center, P.O.
Box 4524, Albuquerque, NM 87106] Vol. 19, No. 2 (Summer, 1994),
pgs. 86-90.]

Medical science has made impressive gains against acute
disease this century, but during the past 20 years it has become
obvious that chronic disease represents a much more difficult
challenge. Throughout the industrialized world, many chronic
diseases are steadily increasing. There is substantial and
growing evidence that these increases are linked to widespread
low-level chemical contamination of the environment. An
aggressive new environmental movement has come into being to
confront these human problems. Here is a brief, incomplete
catalog:

  • The incidence (occurrence) of many kinds of cancer has
    been increasing for 50 years, and continues to increase today;
    particularly steep increases have occurred among cancers of the
    testicles and prostate, the female breast, the brain, and the
    kidney but the incidence of 10 other kinds of cancer has
    relentlessly risen for five decades. [1]

  • The incidence of certain birth defects — among them,
    heart defects, defects of the male genitalia, and facial clefts
    — is rising. [2]

  • Increasingly, couples in their prime reproductive years
    are having trouble reproducing; [3] this may be due in part to a
    50% decline in sperm count that has been documented among men
    worldwide over the past 50 years, a decline that is apparently
    continuing. [4]

  • Certain female reproductive disorders are also
    increasing; the number of ectopic pregnancies is rising, [5] as is
    the prevalence of endometriosis, a painful disease associated
    with the menstrual cycle, which now afflicts 5 million American
    women. In 1960, breast cancer struck one in 20 women; today, it
    strikes one in 9. [6]

  • Certain disorders of the central nervous system — ALS
    [Lou Gehrig’s disease] and Parkinson’s disease — are increasing.
    Some public health leaders suspect chemical contamination is the
    cause. [7]

  • Certain blood disorders are increasing (e.g., multiple
    myeloma, and septicemia). [8]

  • The prevalence of some well-known diseases of the immune
    system, such as asthma and diabetes, is steadily increasing. [9]

  • A new disease has emerged called “multiple chemical
    sensitivity” — a disease characterized by extreme sensitivity
    to low levels of various chemicals, sometimes including odors
    from new carpets, perfumes, and the fragrances in commercial
    products such as waxes and detergents. Symptoms range in
    severity from an itchy rash to coma. [10]

  • Today in the U.S., occupational disease kills an
    estimated 60,000 workers each year. [11] In addition to these
    preventable deaths, workplace conditions cause an estimated
    350,000 new cases of serious illness among workers each year,
    according to Philip J. Landrigan, chairman of the department of
    community medicine at the Mount Sinai School of Medicine in New
    York.

  • The American Academy of Pediatrics estimates that, in any
    given year somewhere between 2 and 4 million American children
    (and approximately 400,000 fetuses) have sufficient lead in
    their blood to diminish their IQ, reduce their physical stature,
    damage their hearing, decrease their hand-eye coordination and
    impair their ability to pay attention in school. [12] This
    damage is thought to be permanent. Lead is a soft, gray toxic
    metal that has been mined from the earth and formed into useful
    items for 5000 years. Its toxicity to miners and workers was
    well established among ancient Greeks and Romans long before the
    birth of Christ.

    This list is not exhaustive but indicates a sea of troubles
    rising, much of it linked to chemical contamination.

    The grass-roots movement for environmental justice, which
    has come into being to confront these problems, has had many
    local victories. But its most far-reaching accomplishment has
    been to force a change in the way we think about controlling
    chemical contamination — from managing pollution to preventing
    pollution. This is a very fundamental shift. Major polluters
    and their acolytes in government are now giving lip service to
    “pollution prevention” but almost no one in the U.S. has yet put
    it into practice to any significant degree. What is most
    important is that the new approach has been spelled out and is
    now available as a standard against which to measure any claims
    of pollution prevention.

    As the American Chemical Society noted in 1993, [13] there
    are two basic philosophies of chemical regulation: for 20 years
    the U.S. has been using a permissive chemical-by-chemical
    approach, which assumes all chemicals are innocent until proven
    guilty, and which demands rigorous scientific proof of harm
    before regulation can occur. We call this the “prove harm”
    philosophy of chemical regulation. This philosophy assumes that
    every ecosystem and every species has some “assimilative
    capacity,” some capacity to absorb harm without suffering
    irreversible damage. This philosophy assumes further that (a)
    scientists can discover the assimilative capacity of every
    ecosystem and every species (“risk assessment” is the technique
    that is supposed to accomplish this); that (b) regulators can
    and will then establish effective limits on the cumulative
    harmful activities so as to avoid irreversible damage; and that
    (c) we already know which substances are harmful and which are
    not; or, in the case of substances that we never suspected are
    harmful, we will be warned of their possible dangers by
    traumatic but sub-lethal shocks that alert us to the danger
    before it is too late. Recent history indicates clearly that
    all three of these assumptions are simply wrong.

    Happily, an alternative philosophy of chemical regulation
    has developed in recent years in Europe and in some obscure
    corners of the U.S. government. These changes have been driven
    by growing citizen demands at the local level, world-wide.

    The new philosophy — which we call clean technology and
    others call clean production or industrial ecology — has been
    articulated most succinctly in 2 reports from the International
    Joint Commission (IJC), the U.S.-and-Canadian governmental body
    in charge of water quality for the Great Lakes.

    In its sixth (1992) and seventh (1994) biennial reports,
    the IJC has described and advocated a pollution-prevention
    approach commensurate with the size and nature of the
    problem. [14]

    Basically, the clean technology approach regulates whole
    classes of chemicals instead of one chemical at a time. Given
    that some 60,000 chemicals are now produced in commercial
    quantities, with 500 to 1000 new ones introduced into commerce
    each year, regulating classes of chemicals is simpler and more
    manageable than the chemical-by-chemical one-at-a-time approach.

    Eliminate Persistent Toxic Substances

    The IJC now recommends defining a class of chemicals called
    “persistent toxic substances,” which should then be eliminated
    because they cannot be managed safely.

    The IJC recommends that a persistent toxic substance be
    defined as any toxic chemical that bioaccumulates (accumulates
    in food chains), or any toxic chemical that has a half-life
    greater than eight weeks in any medium (water, air, sediment,
    soil, or living things). (The half-life is the time it takes
    for half of any substance to degrade and disappear once it has
    been released into the environment.) Toxic substances with
    either of these characteristics should be eliminated, the IJC
    says. [15]

    The IJC takes its definition of a toxic substance from the
    1978 Great Lakes Water Quality Agreement, which has been adopted
    by the federal government of the U.S. and Canada:

    A toxic substance is anything that can “cause death,
    disease, behavioral abnormalities, cancer, genetic mutations,
    physiological or reproductive malfunctions or physical
    deformities in any organism, or its offspring, or which can
    become poisonous after concentrating in the food chain or in
    combination with other substances.”

    A substance bioaccumulates if its concentration increases
    as it moves through the food chain. For example, DDT may be
    found at one ppm (part per million) in fish and at 10 ppm in
    fish- eating birds. Thus DDT bioaccumulates. The IJC says any
    toxic substance that bioaccumulates should be eliminated.

    A substance is defined as persistent if it has a half-life
    greater than 8 weeks in any medium (air, water, soil, sediment,
    or living things). As noted above, the “half life” of a
    substance is the time it takes for half of it to degrade and
    disappear. For example, DDT has a “half-life” of about 59 years
    in temperate climates; if a pound of DDT is released into soil
    today, half of it will still exist 59 years from now. After 10
    half-lives only a small fraction of the original amount exists
    (1/1024), so 10 half-lives is the rule of thumb for how long a
    substance remains in the environment. By this measure, DDT
    remains in the environment for 590 years after it has been
    released. The IJC recommends that any persistent toxic
    substance be eliminated.

    Adopt The Principle of Precautionary Action

    The IJC recommends that the U.S. and Canada adopt the
    principle of precautionary action. The precautionary principle
    says that, to avoid irreparable harm to the environment and to
    human health, precautionary action should be taken wherever it
    is acknowledged that a practice (or substance) could cause harm,
    even without conclusive scientific proof that it has caused harm
    or does cause harm, the practice (or emissions of the substance)
    should be prevented and eliminated. [16]

    Adopt A Weight-of-the-Evidence Approach

    The IJC recommends that the U.S. and Canada adopt a “weight
    of the evidence” approach, not waiting for scientific certainty
    to be established but taking action to protect against toxics as
    soon as the “weight of the evidence” indicates the need for
    action. [17]

    End Reliance on Risk Assessment and Numerical Standards

    In recommending a “weight of the evidence” approach and in
    recommending the elimination of all persistent toxic substances,
    the IJC has turned away from risk assessment and numerical
    standards. [18] Today risk assessment and numerical standards
    form the backbone of the U.S. regulatory philosophy for
    controlling toxic substances. Numerical standards supposedly
    reflect the “assimilative capacity” of ecosystems, and humans.
    Risk assessment is the technique employed to establish numerical
    standards.

    Adopt the Principle of Reverse Onus

    The principle of “reverse onus” says that chemicals should
    be considered guilty until proven innocent, not the other way
    around. Accordingly, the proponents of a chemical’s production
    and use should bear responsibility for proving that a substance
    is not harmful to the environment or human health. Under the
    present system, society at large bears that responsibility and
    regulatory action cannot be taken until specific, widespread
    harm has been conclusively demonstrated and rigorously
    documented. The IJC says, “This principle [of reverse onus]
    should, in the Commission’s view, be adopted for all human-made
    chemicals shown or reasonably suspected to be persistent and
    toxic, including those already manufactured or otherwise in
    commerce.” [19]

    In sum, the IJC said in 1992, “It is clear to us that
    persistent toxic substances have caused widespread injury to the
    environment and to human health. As a society we can no longer
    afford to tolerate their presence in our environment and in our
    bodies…. Hence, if a chemical or group of chemicals is
    persistent, toxic and bioaccumulative, we should immediately
    begin a process to eliminate it. Since it seems impossible to
    eliminate discharges of these chemicals through other means, a
    policy of banning or sunsetting their manufacture, distribution,
    storage, use and disposal appears to be the only
    alternative.” [20] The IJC defines “sunsetting” as “a
    comprehensive process to restrict, phase out, and eventually ban
    the manufacture, generation, use and disposal of a persistent
    toxic substance.” [21]

    The IJC said, “Such a strategy should recognize that all
    persistent toxic substances are dangerous to the environment,
    deleterious to the human condition, and can no longer be
    tolerated in the ecosystem, whether or not unassailable
    scientific proof of acute or chronic damage is universally
    accepted…. Therefore the focus must be on preventing the
    generation of persistent toxic substances in the first place,
    rather than trying to control their use, release, and disposal
    after they are produced.” [22]

    Implementing the new philosophy of chemical control will
    not be easy. Thanks to so-called “free trade” laws,
    corporations are rapidly gaining new powers and new freedoms to
    do as they please, to move anywhere where labor is cheap and
    pollution laws are lax. Governments and democratic institutions
    at all levels are being weakened. Nevertheless, for reasons
    given earlier, the survival of the human species is now in
    doubt; therefore we have no choice but to meet the challenge by
    developing a world-wide grass-roots movement for environmental
    justice, to confront the poisoners everywhere, to force them to
    adopt a real pollution prevention philosophy backed by real
    pollution prevention programs. I have no illusions that it will
    be easy, but I also have no doubt that humanity is up to the
    challenge.
    &nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp&nbsp
    –Peter Montague

    END OF ARTICLE


    SIDEBAR


    We do not know what all of the effects of human
    exposure will be over many years. Future research
    will clarify whether low- level and long-term
    exposures, repeated exposures, or isolated
    short-term exposures at sensitive stages of fetal
    development are most critical. For the Commission,
    however, there is sufficient evidence now to infer
    a real risk of serious impacts in humans.
    Increasingly, human data support this conclusion.

    IMPLICATIONS OF INACTION

    The questions then become: what–if any–risks
    of injury are we as individuals and as a society
    willing to accept? How long can we afford to wait
    before we act? Why take any risks of having such
    potentially devastating results? In this vein, the
    Commission poses a number of other specific but
    very fundamental questions:

  • What if, as current research suggests, the
    startling decrease in sperm count and the alarming
    increase in the incidence of male genital tract
    disorders are in fact caused in part as a result of
    in utero exposure to elevated levels of
    environmental estrogens?

  • What if, as current research suggests, the
    epidemic in breast cancer is a result in part of
    the great numbers and quantities of estrogen-like
    compounds that have been and are being released
    into the environment?

  • What if the documented declining learning
    performance and increasing incidence of problem
    behaviour [sic] in school children are not functions
    of the educational system? What if they are the
    result of exposure to developmental toxicants that
    have been and are being released into the
    children’s and parents’ environment, or to which
    they have been exposed in utero?

    The implications of any of the above questions
    being answered in the affirmative are
    overwhelming. The implications of all of the above
    questions being answered in the affirmative are
    catastrophic, in terms of human suffering and the
    potential liability for that suffering and
    attendant health costs. Mounting evidence points to
    the latter possibility. Surely, there can be no
    more compelling self interest to force us to come
    to grips with this problem than the spectre of
    damaging the integrity of our own species and its
    entire environment.

    Source of this quotation: International Joint
    Commission, Seventh Biennial Report on Great Lakes
    Water Quality (Washington, DC and Ottawa, Ontario:
    International Joint Commission, 1994), pg. 5. This
    report is available free from the International
    Joint Commission, 1250 23rd Street, N.W., Suite
    100, Washington, DC 20440; telephone: (202)
    736-9000.


    End Notes

    [1] See Barry A. Miller and others, editors, Cancer Statistics
    Review 1973-1989 [National Institutes of Health Publication No.
    92-2789] (Bethesda, Md.: National Cancer Institute, 1992). And:
    Devra Lee Davis and Joel Schwartz, “Trends in Cancer Mortality:
    US White Males and Females, 1968-83,” The Lancet Vol. I (March
    19, 1988), pgs. 633-636. And: Devra Lee Davis, David Hoel, John
    Fox, and Alan Lopez, “International Trends in Cancer Mortality
    in France, West Germany, Italy, Japan, England and Wales, and
    the USA,” The Lancet Vol 366, No. 8713 (August 25, 1990), pgs.
    474- 481. And: Devra Lee Davis and David Hoel, editors, Trends
    in Cancer Mortality in Industrial Countries (New York: New York
    Academy of Sciences, 1990). For a discussion, see Tim
    Beardsley, “A War Not Won–Trends in Cancer Epidemiology,”
    Scientific American Vol. 270 (January 1994), pgs. 130-138. And,
    finally, see Devra Davis and others, “Decreasing Cardiovascular
    Disease and Increasing Cancer Among Whites in the United States
    From 1973 Through 1987,” Journal of the American Medical
    Association Vol. 271 (February 9, 1994), pgs. 431-437.

    [2] Larry D. Edmonds and Levy M. James, “Temporal Trends in the
    Prevalence of Congenital Malformations at Birth Based on the
    Birth Defects Monitoring Program, United States, 1979-1987,”
    MMWR [Morbidity and Mortality Weekly Report] CDC [Centers For
    Disease Control] Surveillance Summaries Vol. 39 No. SS-4
    (December, 1990), pgs. 19-23. Increasing birth defects in male
    genitalia are discussed in A. Giwercman and N.E. Skakkebaek,
    “The human testis–an organ at risk?” International Journal of
    Andrology Vol. 15 (1992), pgs. 373-175. And see: Richard M.
    Sharpe and Niels E. Skakkebaek, “Are oestrogens involved in
    falling sperm counts and disorders of the male reproductive
    tract?” The Lancet Vol. 341 (May 29, 1993), pgs. 1392-1395.

    [3] Increasing infertility among Americans in their prime
    reproductive years is discussed in Appendix A, “Reproductive
    Dysfunction in the Population,” pgs. 341-346, in Office of
    Technology Assessment, Reproductive Health Hazards in the
    Workplace [OTA-BA-266] (Washington, D.C.: U.S. Government
    Printing Office, December, 1985).

    [4] Elisabeth Carlsen and others, “Evidence for decreasing
    quality of semen during past 50 years,” British Medical Journal
    Vol. 305 (1992), pgs. 609-613. And see: Richard M. Sharpe and
    Niels E. Skakkebaek, “Are oestrogens involved in falling sperm
    counts and disorders of the male reproductive tract?” The Lancet
    Vol. 341 (May 29, 1993), pgs. 1392-1395; R. M. Sharpe,
    “Declining sperm counts in men –is there an endocrine cause?”
    Journal of Endocrinology, Vol. 136 (1993), pgs. 357-360;
    Constance Holdren, “The Hazards of Estrogens,” Science Vol. 260
    (May 28, 1993), pgs. 1238-1239; “Estrogenic Chemicals May Lower
    Sperm Counts,” C&EN [Chemical & Engineering News] Vol. 71 No. 23
    (June 7, 1993), pg. 28.

    [5] Increases in ectopic pregnancies are documented in MMWR
    [Morbidity and Mortality Weekly Report] CDC Surveillance
    Summaries Vol. 39 No. SS-4 (December, 1990), pgs. 9-19.

    [6] Bette Hileman, “Environmental Estrogens Linked to
    Reproductive Abnormalities, Cancer,” C&EN [Chemical &
    Engineering News] Vol. 72 No. 5 (January 31, 1994), pgs. 19-23.
    David E. Larson, editor, Mayo Clinic Family Health Book (N.Y.:
    William Morrow, 1990), pgs. 1101-1102. Sherry E. Rier and
    others, “Endometriosis in Rhesus Monkeys (Macaca mulatta)
    Following Chronic Exposure to
    2,3,7,8-Tetrachlorodibenzo-P-dioxin,” Fundamental and Applied
    Toxicology Vol. 21 (1993), pgs. 433-441.

    [7] Increases in Parkinson’s disease and in amyotrophic lateral
    sclerosis (ALS, or Lou Gehrig’s disease) are documented in
    Office of Technology Assessment, Neurotoxicity; Identifying and
    Controlling Poisons of the Nervous System [OTA-BA-436]
    (Washington, D.C.: U.S. Government Printing Office, April,
    1990); see, for example, Figures 2-2 and 2-3 on pg. 55.

    [8] Increases in multiple myeloma are discussed in Jack Cuzick,
    Ramon Velez, and Richard Doll, “International Variations and
    Temporal Trends in Mortality from Multiple Myeloma,”
    International Journal of Cancer Vol. 32 (1983), pgs. 13-19,
    which was updated in Jack Cuzick, “International Time Trends for
    Multiple Myeloma,” in Devra Lee Davis and David Hoel, editors,
    Trends in Cancer Mortality in Industrial Countries (New York:
    New York Academy of Sciences, 1990), pgs. 205-214. Increases in
    septicemia are documented in MMWR [Morbidity and Mortality
    Weekly Report] Vol. 39 No. 2 (January 19, 1990), pg. 31-34.

    [9] A. Sonia Buist and William M. Vollmer, “Reflections on the
    Rise in Asthma Morbidity and Mortality,” Journal of The American
    Medical Association October 3, 1990, pgs. 1719-1720. And: Peter
    J. Gergen and others, “National Survey of the Prevalence of
    Asthma Among Children in the United States, 1976 to 1980,”
    Pediatrics Vol. 81 (1988), pgs. 1-7. And: Kevin B. Weiss and
    Diana K. Wagener, “Changing Patterns of Asthma Mortality,”
    Journal of The American Medical Association Vol. 264 (1990),
    pgs. 1683-1687. And Richard Evans III and others, “National
    Trends in the Morbidity and Mortality of Asthma in the U.S.,”
    Chest Vol. 91 No. 6 (June 1987) Supplement, pgs. 65S-74S.

    [10] See Board on Environmental Studies and Toxicology,
    National Research Council, Multiple Chemical Sensitivities
    (Washington, D.C.: National Academy Press, 1992). Multiple
    chemical sensitivity–an adverse reaction to low levels of many
    different chemicals with symptoms that range from sniffles to
    coma–afflicts 10% to 15% of the American public, and appears to
    be increasing, says Bette Hileman, “Multiple Chemical
    Sensitivity,” C&EN [Chemical & Engineering News] Vol. 69 No. 29
    (July 22, 1991), pg. 34. Hileman says, “The lack of a clear
    definition or diagnostic test for MCS [multiple chemical
    sensitivity] makes it very difficult to estimate its prevalence.
    However there is much indirect evidence that the number of
    people diagnosed with MCS is increasing.” This emerging disease
    has been subject of two excellent book-length studies: In 1990
    the New Jersey Department of Health published a report by
    Nicholas Ashford and Claudia Miller, Chemical Sensitivity, which
    is distributed by National Center for Environmental Health
    Strategies (NCEHS), 1100 Rural Ave., Voorhees, NJ 08043; phone
    (609) 429-5358. $17.00. See also Nicholas Ashford and Claudia
    Miller, Chemical Exposures: Low Levels And High Stakes (New
    York: Van Nostrand Reinhold, 1990).

    [11] “Studies conducted in New York state have estimated that
    50,000 to 70,000 workers die each year from chronic occupational
    diseases resulting from past exposures to toxic substances,”
    writes Philip Landrigan of Mount Sinai School of Medicine in New
    York City. “Included are lung cancers and mesothelioma [cancer
    of the lining of the chest cavity] from asbestos exposure;
    bladder cancer among dye workers; leukemia and lymphoma in
    workers exposed to benzene and ionizing radiation; chronic
    bronchitis in workers exposed to dusts; disorders of the nervous
    system (including possibly dementia, Parkinson’s disease, and
    motoneuron disease [Lou Gehrig’s disease]) in workers exposed to
    pesticides, solvents, and certain other neurotoxins; renal
    [kidney] failure in workers exposed to lead; and cardiovascular
    disease in workers exposed to carbon monoxide and carbon
    disulfide.” See Philip J. Landrigan, “Commentary: Environmental
    Disease–A Preventable Epidemic,” American Journal of Public
    Health Vol. 82 (July 1992), pgs. 941-943.

    [12] Committee on Environmental Health, American Academy of
    Pediatrics, “Lead Poisoning: From Screening to Primary
    Prevention,” Pediatrics Vol. 92 (July 1993), pgs. 176-183.

    [13] Bette Hileman, “Concerns Broaden over Chlorine and
    Chlorinated Hydrocarbons,” C&EN [Chemical & Engineering News]
    Vol. 71, No. 16 (April 19, 1993), pg. 20.

    [14] International Joint Commission, Sixth Biennial Report
    Under the Great Lakes Water Quality Agreement of 1978 To The
    Governments of the United States and Canada and the State and
    Provincial Governments of the Great Lakes Basin (Washington,
    D.C., and Ottawa, Ontario, 1992) and International Joint
    Commission, Seventh Biennial Report Under the Great Lakes Water
    Quality Agreement of 1978 To The Governments of the United
    States and Canada and the State and Provincial Governments of
    the Great Lakes Basin (Washington, D.C., and Ottawa, Ontario,
    1994). Both
    reports are indispensable reading and are available free from
    International Joint Commission, 1250 23rd Street, N.W., Suite
    100, Washington, DC 20440; telephone: (202) 736-9000.

    [15] International Joint Commission, Sixth Biennial Report
    (cited above), pgs. 4, 57.

    [16] The principle of precautionary action did not originate
    with the IJC. The Ministerial Declaration of the Second
    International Conference on the Protection of the North Sea,
    held in London November 24-25, 1987, contains the following
    language: “[We agree to] accept the principle of safeguarding
    the marine ecosystem of the North Sea by reducing polluting
    emissions of substances that are persistent, toxic, and liable
    to bioaccumulate at source by the best available technology and
    other appropriate measures. This applies especially where there
    is reason to assume that certain damage or harmful effects on
    the living resources of the sea are likely to be caused by such
    substances, even where there is no scientific evidence to prove
    a causal link between emissions and effects (‘the principle of
    precautionary action’);”

    The United Nations Environment Program (UNEP) Governing
    Council on May 25, 1989, adopted Decision 15/27 urging all
    governments to adopt “the ‘principle of precautionary action’ as
    the basis of their policy with regard to the prevention and
    elimination of marine pollution.” Part of the rationale given
    was “that waiting for scientific proof regarding the impact of
    pollutants discharged into the marine environment may result in
    irreparable damage to the marine environment and in human
    suffering.”

    [17] International Joint Commission, Sixth Biennial Report
    (cited above), pgs. 22-23.

    [18] International Joint Commission, Seventh Biennial Report
    (cited above), pg. 28.

    [19] International Joint Commission, Seventh Biennial Report
    (cited above), pg. 9.

    [20] International Joint Commission, Sixth Biennial Report
    (cited above), pg. 4.

    [21] International Joint Commission, Sixth Biennial Report
    (cited above), pg. 25.

    [22] International Joint Commission, Sixth Biennial Report
    (cited above), pgs. 5, 25.